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977umb_Becker.qxd
Using lymphocytes from nine unrelated patients
with multiple symmetric lipomatosis we investigated
a possible defect in the mitochondrial respiratory
chain as the biochemical cause for the disease. A
significant decrease in oxygen consumption of intact
lymphocytes as well as a decreased activity of the individual
components of the respiratory chain were detected.
These findings are consistent with the recently
described deletions and point mutations of mitochondrial
DNA in patients suffering from this disease.
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